Testosteron propionat von ip

Testosterone Propionate also converts to both Estrogen (through Aromatization) and Dihydrotestosterone (through 5a-reduction). Most of the side effects people experience with testosterone use is actually from it’s conversion to these two substrates. Thus, hair loss , water retention, acne, and other side effects are possible with use of this drug. Conversion to these hormones is also responsible for some of testosterone’s ability to build muscle; therefore when many side effects are avoided with the use of ancillary compounds, some of the muscle building properties are also stunted.

Testosterone propionate was introduced in 1937 by Schering AG in Germany under the brand name Testoviron . [7] It was the first ester of testosterone to be introduced, [8] and was the major form of testosterone used medically before 1960. [7] In the 1950s, longer-acting testosterone esters like testosterone enanthate and testosterone cypionate were introduced and superseded testosterone propionate. [8] Although rarely used nowadays due to its short duration, [9] testosterone propionate remains medically available and is still marketed in the United States . [7] [10]

For the Testosterone Replacement Therapy candidate generally dosing will fall in the  100mg-200mg per week  range and rarely surpassing this amount. However, as frequent injections are necessary most physicians will generally opt for larger ester forms, most commonly Testosterone-Cypionate so the patient only has to inject once per week.

For the performance enhancer the dosing will be much larger and the varying levels will be much wider too but he must necessarily inject frequently as discussed if he is going to maintain stability. Generally the minimal dose when supplementing for performance purposes will be 100mg every other day with  200mg every other day  being as high as most will ever go. Yes, you an absolutely take more, there is no set in stone amount but understand such high-end doses can open up the doors to side-effects that you may have wished to remain shut. In elite level competitive sports, especially in elite level bodybuilding it is not uncommon to see doses surpass a 2,000mg per week range but this can be very harsh and there is simply no way such a dosing could ever be recommended in a responsible manner.

As with all cycles, as natural testosterone production will be suppressed, once use is discontinued and responsible use will include breaks Post Cycle Therapy (PCT) is a must in order to aid and speed up the recovery process. This is where Testosterone-Propionate has a bit of advantage over many other forms of testosterone; as a small ester that clears very quickly if your cycle ends with Testosterone-Propionate and there is no other long ester gear present in your system your PCT plan can start almost immediately. Remember, the sooner we start our PCT the better off we’ll be and such rapid PCT application cannot be accomplished with long ester based testosterone. If your cycle ends with Testosterone-Propionate you could reasonably wait 3 days before PCT therapy begins; if it ends with large ester gear you will need to wait approximately 2 weeks.

In androgen-responsive target tissues such as the skin, scalp, and prostate, the high relative androgenicity of testosterone is dependant on its reduction to dihydrotestosterone (DHT). The 5-alpha reductase enzyme is responsible for this metabolism of testosterone. The concurrent use of a 5-alpha reductase inhibitor such as finasteride or dutasteride will interfere with site-specific potentiation of testosterone action, lowering the tendency of testosterone drugs to produce androgenic side effects. It is important to remember that both anabolic and androgenic effects are mediated via the cytosolic androgen receptor. Complete separation of testosterone’s anabolic and androgenic properties is not possible, even with total 5-alpha reductase inhibition.

Testosteron propionat von ip

testosteron propionat von ip

In androgen-responsive target tissues such as the skin, scalp, and prostate, the high relative androgenicity of testosterone is dependant on its reduction to dihydrotestosterone (DHT). The 5-alpha reductase enzyme is responsible for this metabolism of testosterone. The concurrent use of a 5-alpha reductase inhibitor such as finasteride or dutasteride will interfere with site-specific potentiation of testosterone action, lowering the tendency of testosterone drugs to produce androgenic side effects. It is important to remember that both anabolic and androgenic effects are mediated via the cytosolic androgen receptor. Complete separation of testosterone’s anabolic and androgenic properties is not possible, even with total 5-alpha reductase inhibition.

Media:

testosteron propionat von iptestosteron propionat von iptestosteron propionat von iptestosteron propionat von iptestosteron propionat von ip

http://buy-steroids.org